講演・口頭発表等 - 岡澤　均

41. Kanoh Kondo, Xigui Chen, Kazumi Motoki, Hidenori Homma, Hitoshi Okazawa. In vivo imaging of intracellular organelle in neurons of Alzheimer's disease and other neurodegenerative disease model. The 41st Annual Meeting of the Japan Neuroscience Society 2018.07.27 Kobe Convention Center （Hyogo Kobe）

42. Hitoshi Okazawa. Comprehensive analyses reveal new concepts of neurodegeneration in time axis. The 41st Annual Meeting of the Japanese Neuroscience Society 　JNS-JSN Joint Symposium 2018.07.26 Kobe Convention Center （Hyogo Kobe）

43. Hitoshi Okazawa. New Molecularly Targeted Therapeutics Deduced from Unbiased Approaches. 21st US-Japan Cellular and Gene Therapy Conference 2018.03.01 FDA White Oak Campus(Maryland, USA)

44. Hitoshi Okazawa. Common pathologies across multiple neurodegenerative diseases revealed from unbiased approaches. 12th International Symposium of the Institute Network 2017.11.28 The Institute of Medical Science, The University Of Tokyo(Tokyo)

45. Hitoshi Okazawa. HMGB1 and Neurodegeneration. 8th International DAMPs and Alarmins Symposium (iDEAs) 2017.11.06 Cold Spring Harbor Laboratory （NewYork, USA）

46. Hitoshi Okazawa. Conceptual merge between aging and neurodegeneration. 16th Surugadai International Symposium & Joint Usage/Research Program of Medical Research Institute International Symposium 2017.10.11 Tokyo Medical and Dental University(Tokyo)

47. Hitoshi Okazawa. Gene Therapy Against Spinocerebellar Ataxia. XXIII World Congress of Neurology (WCN2017） 2017.09.19 Kyoto International Conference Center (Kyoto)

48. Juliana Bosso Taniguchi, Kanoh Kondo, Kyota Fujita, Xigui Chen, Hidenori Homma , Ying Mao , Kazuhiko Tagawa , Hitoshi Okazawa. RPA1 AMELIORATES SYMPTOMS OF MUTANT ATAXIN-1 KNOCK-IN MICE AND ENHANCE DNA DAMAGE REPAIR. XXIII World Congress of Neurology (WCN 2017) 2017.09.17 Kyoto International Conference Center (Kyoto)

49. Kyota Fujita, Kazumi Motoki, Kazuhiko Tagawa, Xigui Chen, Hiroshi Hama, Kazuyuki Nakajima, Hidenori Homma, Takuya Tamura, Atsushi Miyawaki, Hitoshi Okazawa. HMGB1 TRIGGERS NEURITE DEGENERATION VIA TLR4-MARCKS, AND IS A POTENTIAL THERAPEUTIC TARGET FOR ALZHEIMER’S DISEASE. XXIII World Congress of Neurology (WCN 2017) 2017.09.17 Kyoto International Conference Center (Kyoto)

50. Hitoshi Okazawa. Common pathologies across multiple neurodegenerative diseases revealed from unbiased approaches. MyNEURO 2017 2017.08.13 Hotel Istana（KualaLumpur, Malaysia）

51. Xigui Chen, Mao Ying, Xu Min, Kyota Fujita, Kazumi Motoki, Toshikazu Sasabe, Hidenori Homma, Miho Murata, Kazuhiko Tagawa, Takuya Tamura, Kaye Julia, Steven Finkbeiner, Giovanni Blandino, Marius Sudol, Kanoh Kondo, Hitoshi Okazawa. Targeting TEAD/YAP-transcription-dependent necrosis, TRIAD, ameliorates Huntington's disease pathology. The 40th Annual Meeting of the Japan Neuroscience Society 2017.07.22 Kyoto International Conference Center (Kyoto)

52. Masaki Sone, Koto Furotani, Taka-aki Yajima, Takuya Tamura, Hitoshi Okazawa. Effect of specific inhibition of synaptic delivery of APP by loss-of-function of yata on the Drosophila Alzheimer's disease model. The 40th Annual Meeting of the Japan Neuroscience Society 2017.07.21 Makuhari Messe(Chiba)

53. Kyota Fujita, Kazumi Motoki, Kazuhiko Tagawa, Xigui Chen, Hiroshi Hama, Kazuyuki Nakajima, Hidenori Homma, Takuya Tamura, Masahisa Katsuno, Takashi Saito, Takaomi Saido, Gen Sobue, Atsushi Miyawaki, Hitoshi Okazawa. HMGB1, a pathogenic molecule that induces neurite degeneration via TLR4-MARCKS, is a potential therapeutic target for Alzheimer's disease. The 40th Annual Meeting of the Japan Neuroscience Society 2017.07.20 Makuhari Messe(Chiba)

54. Juliana Bosso Taniguchi, Kanoh Kondo, Kyota Fujita, Chen Xigui, Hidenori Homma, Takeaki Sudo,Ying Mao, Kei Watase, Toshihiro Tanaka, Kazuhiko Tagawa, Takuya Tamura, Shin-Ichi Muramatsu, Hitoshi Okazawa. RpA1 ameliorates symptoms of mutant ataxin-1 knock-in mice and enhances DNA damage repair. The 40th Annual Meeting of the Japan Neuroscience Society 2017.07.20 Makuhari Messe(Chiba)

55. Hitoshi Okazawa. Impaired DNA Damage Repair in SCA1 and Other CAG Repeat Diseases. Gordon Research Conference - CAG Triplet Repeat Disorders 2017.06.06 Mount Snow Grand Summit Hotel （Vermont, USA）

56. Furotani K, Yajima T, Toyokawa M, Nakayama M, Tamura T, Okazawa H, Sone M. Effect of inhibition of synaptic delivery of APP by loss-of-function of yata for the Drosophila Alzheimer's disease model. The 39th Annual Meeting of the Molecular Biology Society of Japan 2016.12.01 Rikkyo University (Tokyo)

57. Okazawa H. Therapeutics development of Alzheimer’s disease targeting on Phase 0. 15th Surugadai International Symposium 2016.11.29 Tokyo Medical and Dental University (Tokyo)

58. Furotani K, Yajima T, Toyokawa M, Nakayama M, Tamura T, Okazawa H, Sone M. Effect of inhibition of synaptic delivery of APP by loss-of-function of yata for the Drosophila Alzheimer's disease model. The 12th Japanese Drosophila Research Conference 2016.09.09 Rikkyo University (Tokyo)

59. Mizuguchi M, Nabeshima Y, Obita T, Okazawa H. Interaction of spliceosomal proteins PQBP1, U5-15kD, and WBP11. The XXVIIth International Conference on Magnetic Resonance in Biological Systems 2016.08.22 International Conference Center Kyoto (Kyoto)

60. Okazawa H. Gene therapy to conquer spinocerebellar ataxia. The 22nd Annual Meeting of Japan Society of Gene and Cell Therapy 2016.07.30 Toranomon Hills Forum (Tokyo)