Personnel Information

写真a

UEDA Mahoko


Job title

Assistant Professor

Graduating School 【 display / non-display

  • Hiroshima University, Faculty of Science, Graduated

Graduate School 【 display / non-display

  • The Graduate University for Advanced Studies, Doctor's Course, Completed

Campus Career 【 display / non-display

  • 2020.07
    -
    Now
    Tokyo Medical and Dental University, Medical Research Institute, Medical Genomics, , Assistant Professor

Research Areas 【 display / non-display

  • Genome biology

 

Published Papers & Misc 【 display / non-display

  • Kosugi Y, Uriu K, Suzuki N, Yamamoto K, Nagaoka S, Kimura I, Konno Y, Aso H, Willett BJ, Kobayashi T, Koyanagi Y, Ueda MT, Ito J, Sato K. Comprehensive Investigation on the Interplay between Feline APOBEC3Z3 Proteins and Feline Immunodeficiency Virus Vif Proteins. Journal of virology. 2021.06; 95 (13): e0017821. ( PubMed, DOI )

  • Matsuzawa Ayumi, Lee Jiyoung, Nakagawa So, Itoh Johbu, Takahashi Ueda Mahoko, Mitsuhashi Satomi, Kochi Yuta, Kaneko-Ishino Tomoko, Ishino Fumitoshi. HERV-Derived Ervpb1 Is Conserved in Simiiformes, Exhibiting Expression in Hematopoietic Cell Lineages Including Macrophages INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES. 2021.05; 22 (9): ( DOI )

  • Chikuma Shunsuke, Yamanaka Soichiro, Nakagawa So, Ueda Mahoko Takahashi, Hayabuchi Hodaka, Tokifuji Yukiko, Kanayama Masashi, Okamura Tadashi, Arase Hisashi, Yoshimura Akihiko. TRIM28 Expression on Dendritic Cells Prevents Excessive T Cell Priming by Silencing Endogenous Retrovirus JOURNAL OF IMMUNOLOGY. 2021.04; 206 (7): 1528-1539. ( PubMed, DOI )

  • Chikuma S, Yamanaka S, Nakagawa S, Ueda MT, Hayabuchi H, Tokifuji Y, Kanayama M, Okamura T, Arase H, Yoshimura A. TRIM28 expression on dendritic cells prevents excessive T cell priming by silencing endogenous retrovirus Journal of Immunology. 2021; in press

  • Ueda Mahoko Takahashi, Kryukov Kirill, Mitsuhashi Satomi, Mitsuhashi Hiroaki, Imanishi Tadashi, Nakagawa So. Comprehensive genomic analysis reveals dynamic evolution of endogenous retroviruses that code for retroviral-like protein domains MOBILE DNA. 2020.09; 11 (1): 29. ( PubMed, DOI )

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