Personnel Information

写真a

TANIGUCHI JUNKO


Job title

URA

Graduate School 【 display / non-display

  • Chiba University, Doctor's Course, Completed

Campus Career 【 display / non-display

  • 2013.09
    -
    2014.08
    Tokyo Medical and Dental University, Medical Research Institute, Pathophysiology, Neuropathology, Project Assistant Professor
  • 2022.12
    -
    2023.02
    Tokyo Medical and Dental University, Research University Promotion Organization, Research Administration Division, URA
  • 2023.03
     
     
    Tokyo Medical and Dental University, -, -, URA
  • 2023.04
    -
    2024.09
    Tokyo Medical and Dental University, -, -, URA
  • 2024.10
    -
    Now
    Institute of Science Tokyo, -, Institute of Science Tokyo Hospital, Health Science Research and Development Center, URA

External Career 【 display / non-display

  • , Project Researcher
  • , Project Researcher
  • , Project Researcher

Research Areas 【 display / non-display

  • Neuroscience-general

Qualification Acquired 【 display / non-display

  • Administrative Scrivener

 

Published Papers & Misc 【 display / non-display

  1. AgIn: measuring the landscape of CpG methylation of individual repetitive elements 2016.10; 32 (19): 2911-2919. ( PubMed, DOI )

  2. Long-term effects of cerebral hypoperfusion on neural density and function using misery perfusion animal model 2016.04; 6 25072. ( PubMed, DOI )

  3. [C-11]Raclopride binding in the striatum of minimally restrained and free-walking awake mice in a positron emission tomography study 2015.12; 69 (12): 600-606. ( PubMed, DOI )

  4. Modulation of the kallikrein/kinin system by the angiotensin-converting enzyme inhibitor alleviates experimental autoimmune encephalomyelitis 2014.11; 178 (2): 245-252. ( PubMed, DOI )

  5. Changes in cortical microvasculature during misery perfusion measured by two-photon laser scanning microscopy 2014.08; 34 (8): 1363-1372. ( PubMed, DOI )

  6. Evaluation of Rho-Kinase Activity in Mice Brain Using N-[C-11]Methyl-hydroxyfasudil with Positron Emission Tomography 2014.06; 16 (3): 395-402. ( PubMed, DOI )

  7. High-mobility Group Box 1 as A Possible Therapeutic Target for Neuromyelitis Optica and Multiple Sclerosis 2014.06; 20 (7): 908-909.

  8. Cerebral hemodynamic response to acute hyperoxia in awake mice 2014.04; 1557 155-163. ( PubMed, DOI )

  9. Microvascular sprouting, extension, and creation of new capillary connections with adaptation of the neighboring astrocytes in adult mouse cortex under chronic hypoxia 2014.02; 34 (2): 325-331. ( PubMed, DOI )

  10. THE SUBTHALAMIC ACTIVITY AND STRIATAL MONOAMINE ARE MODULATED BY SUBTHALAMIC STIMULATION 2014.02; 259 43-52. ( PubMed, DOI )

  11. Hemodynamic changes during neural deactivation in awake mice: A measurement by laser-Doppler flowmetry in crossed cerebellar diaschisis 2013.11; 1537 350-355. ( PubMed, DOI )

  12. Retinol palmitate prevents ischemia-induced cell changes in hippocampal neurons through the Notch1 signaling pathway in mice 2013.09; 247 182-187. ( PubMed, DOI )

  13. Anti-high mobility group box 1 monoclonal antibody ameliorates experimental autoimmune encephalomyelitis 2013.04; 172 (1): 37-43. ( PubMed, DOI )

  14. Multiple angiogenetic factors are upregulated in POEMS syndrome 2013.01; 92 (2): 245-248. ( PubMed, DOI )

  15. Hypoxia-Induced Cerebral Angiogenesis in Mouse Cortex with Two-Photon Microscopy 2013; 789 15-20. ( PubMed, DOI )

  16. RGMa modulates T cell responses and is involved in autoimmune encephalomyelitis 2011.04; 17 (4): 488-U129. ( PubMed, DOI )

  17. Cytokine and chemokine profiles in neuromyelitis optica: significance of interleukin-6 2010.12; 16 (12): 1443-1452. ( PubMed, DOI )

  18. Chronic Inflammatory Demyelinating Polyneuropathy Sera Inhibit Axonal Growth of Mouse Dorsal Root Ganglion Neurons by Activation of Rho-Kinase 2009.11; 66 (5): 694-697. ( PubMed, DOI )

  19. The effects of age, gender, and body mass index on amplitude of sensory nerve action potentials: Multivariate analyses 2009.09; 120 (9): 1683-1686. ( PubMed, DOI )

  20. LIM-only protein 4 interacts directly with the repulsive guidance molecule A receptor Neogenin 2008.10; 107 (2): 418-431. ( PubMed, DOI )

  21. Neogenin regulates neuronal survival through DAP kinase 2008.10; 15 (10): 1593-1608. ( PubMed, DOI )

  22. BMP inhibition enhances axonal growth and functional recovery after spinal cord injury 2008.05; 105 (4): 1471-1479. ( PubMed, DOI )

  23. Myosin IIA is required for neurite outgrowth inhibition produced by repulsive guidance molecule 2008.04; 105 (1): 113-126. ( PubMed, DOI )

  24. Rap1 is involved in the signal transduction of myelin-associated glycoprotein 2008.02; 15 (2): 408-419. ( PubMed, DOI )

  25. Rho-kinase inhibition enhances axonal regeneration after peripheral nerve injury 2006.09; 11 (3): 217-224. ( PubMed, DOI )

  26. Delayed treatment with Rho-kinase inhibitor does not enhance axonal regeneration or functional recovery after spinal cord injury in rats 2006.08; 200 (2): 392-397. ( PubMed, DOI )

  27. Neogenin mediates cell death through DAP kinase and Rac1 2006; 55 S120.

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Conference Activities & Talks 【 display / non-display

  1. 藤田 幸, 谷口 順子, 内川 昌則, 遠藤 光晴, 羽田 克彦, 久保 武一, 山下 俊英. neogeninの細胞死誘導の分子メカニズム(The mechanism of neogenin induced neuronal cell death). 神経化学 2008.08.01

  2. Taniguchi Junko, Fujitani Masashi, Endo Mitsuharu, Kubo Takekazu, Fujitani Mayumi, Hata Katsuhiko, Yamaguchi Atsushi, Miller Freda D., Kaplan David R., Yamashita Toshihide. ミエリン関連糖蛋白質(MAG)のシグナル伝達におけるRap1の関与(Rap1 is involved in the signal transduction of myelin-associated glycoprotein). 神経化学 2007.08.01

  3. Taniguchi Junko, Hata Katsuhiko, Fujitani Masashi, Kubo Takekazu, Yamashita Toshihide. NeogeninはDAP kinase及びRac1を通じて細胞死を介在する(Neogenin mediates cell death through DAP kinase and Racl). Neuroscience Research 2006.07.01