論文・総説 - 井関　祥子

121. De novo Alu element insertions in FGFR2 identify a distinct pathological basis for Apert syndrome.（共著） American Jounal of Human Genetics. 1999; 64 446-461.

122. Fgfr and osteopontin domians in the developing skull vault are mutually exclusive and can be altered by locally applied FGF2.(jointly worked) Dvelopment . 1997; 124 3375-3384.

123. Fgfr2 and osteopontin domians in the developing vault are mutually exclusive and can be altered by locally applied FGF2.（共著） Development. 1997; 124 3375-3384.

124. Fgfr2 and Osteopontin domains in the developing skull vault are mutually exclusive and can be altered by locally applied FGF2. (jointly worked) Development. 1997; 124 3375-3384.

125. Expression of the Fgfr2 gene during mouse limb and skull development. (jointly worked) Journal of Anatomy. 1995; 188 223-224.

126. A mutation in the Pax-6 gene in rat small eye is Associated with impaired migration of midbraain crest ccells.(jointly worked) Nature Genetics. 1993; 3 299-304.

127. A mutation in the Pax-6 gene in rat small eye is Associated with impaired migration of midbraain crest ccells.(jointly worked) Nature Genetics. 1993; 3 299-304.

128. A mutation in the Pax-6 gene in rat small eye is associated with impaired migration of midbrain crest cells. (jointly worked) Nature Genetics. 1993; 3 299-304.

129. A mutation in the Pax-6 gene in rat small eye is Associated with impaired migration of midbrain crest cells.（共著） Nature Genetics . 1993; 3 299-304.