Personnel Information

写真a

IKEDA Masaaki


Job title

Associate Professor

Laboratory Address

1-5-45 Yushima, Bunkyo-ku, Tokyo 113-8549, Japan

Laboratory Phone number

+81-3-5803-5579

Laboratory Fax number

+81-3-5803-0213

Mail Address

mikeda.emb@tmd.ac.jp

Homepage URL

http://www.tmd.ac.jp/grad/emb/emb-J.htm

Research Areas, Keywords

tumor suppession, cell cycle, transcription, tissue regeneration, chromatin regulation, apoptosis, p300, p53, Rb, E2F

Graduating School 【 display / non-display

  • Tokyo Medical and Dental University, Faculty of Dentistry, 1983, Graduated

Graduate School 【 display / non-display

  • Tokyo Medical and Dental University, Graduate School, Division of Dental Research, Doctor's Course, 1987, Completed

Degree 【 display / non-display

  • Ph.D., Tokyo Medical and Dental University

Campus Career 【 display / non-display

  • 1987.09
    -
    2018.03
    Tokyo Medical and Dental University, Graduate School of Medical and Dental Sciences, Medical and Dental Sciences, Maxillofacial and Neck Reconstruction, Molecular Craniofacial Embryology, Associate Professor
  • 2018.04
    -
    Now
    Tokyo Medical and Dental University, Graduate School of Medical and Dental Sciences, Medical and Dental Sciences, Maxillofacial and Neck Reconstruction, Molecular Craniofacial Embryology, Associate Professor

External Career 【 display / non-display

  • 1987-1988,
  • Research Associate, Section of Craniofacial Development and Anomalies, Institute of Stomatognathic Science, Faculty of Dentistry, Tokyo Medical and Dental University, Tokyo, Japan,
  • 1988-1992,
  • Research Associate, Department of Oral Microbiology, Faculty of Dentistry, Tokyo Medical and Dental University, Tokyo, Japan,
  • 1991-1994,

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Research Areas 【 display / non-display

  • Molecular biology

  • Cell biology

  • Functional basic dentistry

  • Pathobiological dentistry/Dental radiology

 

Research Theme 【 display / non-display

  • Regenaration of hard tissue using Mesenchymal stem cells, 2012.04 - Now

  • Direct conversion of human fibroblasts into osteoblasts and chondrocytes, 2008.04 - Now

Published Papers & Misc 【 display / non-display

  • Saadat KASM, Lestari W, Pratama E, Ma T, Iseki S, Tatsumi M, Ikeda MA. Distinct and overlapping roles of ARID3A and ARID3B in regulating E2F‑dependent transcription via direct binding to E2F target genes. International journal of oncology. 2021.04; 58 (4): ( PubMed, DOI )

  • Arman Kaifee, Saadat Khandakar A. S. M., Igci Yusuf Z., Bozgeyik Esra, Ikeda Masa-Aki, Cakmak Ecir A., Arslan Ahmet. Long noncoding RNA ERICD interacts with ARID3A via E2F1 and regulates migration and proliferation of osteosarcoma cells CELL BIOLOGY INTERNATIONAL. 2020.08; 11434 ( PubMed, DOI )

  • 3. Bozgeyik E, Saadat KASM, Arman K, Bozgeyik I, Ikeda MA. Enhanced E2F1 activity increases invasive and proliferative activity of breast cancer cells through non-coding RNA CDKN2B-AS1 Meta Gene. 2020.06; 24 100691. ( DOI )

  • 2. Saadat KASM, Bozgeyik E, Arman K, Bozgeyik I, Ikeda MA. ARID3A-mediated modulation of TP73 and TP73-AS1 in osteosarcoma cells Gene Reports. 2020.04; 20 100683. ( DOI )

  • Nobuo Tsuchida, Masa-Aki Ikeda, Υoshizumu Ιshino, Michele Grieco, Giancarlo Vecchio. FUCA1 is induced by wild-type p53 and expressed at different levels in thyroid cancers depending on p53 status. International Journal of Oncoloy. 2017.06; 50 (6): 2043-2048. ( PubMed, DOI )

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Books etc 【 display / non-display

  • Ichwan SJA, Bakhtiar MT, Ohtani K, Ikeda MA. Therapeutic Targeting of p53-Mediated Apoptosis Pathway in Head and Neck Squamous Cell Carcinomas:. 2012.02

  • Distinct transcriptional regulation by E2F in cell growth and tumor suppression. "Control of cellular physiology by the transcription factor E2F" p91-105. Research Signpost, 2008 (ISBN : 978-81-308-0230-5)

  • Collaborative Transformation with Two Oncogenes; myc Colloaborating with v-src in primary Cells & with an Immortalization-Positive SV40 Mutated Oncogene in Established rat Cells. "Recent Progress of Life Science Technology in Japan" p119-136. Academic Press, 1989

Conference Activities & Talks 【 display / non-display

  • Khandakar A.S.M. Saadat, Widya Lestari, Teng Ma, Endrawan Pratama, Sachiko Iseki, Kiyoshi Ohtani, Masa-Aki Ikeda. ARID3B modulates E2F target gene expression and cell proliferation.. 2019年度生命科学系学会合同年次大会 2019.12.04 福岡

  • Saadat K ASM, Ikeda MA. Gene-Silencing Efficiency Determination of ARID3B in Osteosarcoma Cell Lines by siRNA and shRNA. 6th International Multidisciplinary Studies Congress 2019.04.26 Gaziantep, Turkey

  • Saadat K ASM, Ikeda MA. Gene-Silencing Efficiency Determination of ARID3B in Osteosarcoma Cell Lines by siRNA and shRNA. III. International Eurasia Multidisciplinary Studies Congress 2019.04.04 Gaziantep, Turkey

  • Öğr. Üyesi. Saadat K ASM. Saadat, Arman K, Ikeda MA. ARID3A Regulates the Tumor Suppressor Tap73 in Osteosarcoma. 2nd ZEUGMA International Congress on Multidisciplinary Studies 2019.01.20 Gaziantep Turkey

  • Saadat K ASM, Arman K, Ikeda MA. ARID3A Regulates the Tumor Suppressor Tap73 in Osteosarcoma. 2nd ZEUGMA International Congress on Multidisciplinary Studies 2019.01.18 Gaziantep Turkey

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